tag:blogger.com,1999:blog-89194034637154150492024-03-19T21:52:18.088+02:00SA Dermatology - Skin Disease Review for Africa by Dr Yacoob Omar CarrimAcne, Atopic Eczema, Cryosurgery, The Elderly, Flexural Rashes, The Infant Skin, Leg Ulcers, Pigmentary Disorders, Bullous Disorders, Psoriasis,Sex,Skin Surgery, Systemic Disease, Urticaria, HIV, Contact Dermatitis,Drug Eruptions, Facial Rashes, Hair Problems,Nail Problems,Insects,The Pregnant Patient,Infections,Skin Cancer (Pigmented Lesions,Melanoma, Non-Melanoma), Sun, Photodermatoses, PDT, Topical Therapy, Tropical Skin Disease & VasculitisUnknownnoreply@blogger.comBlogger26125tag:blogger.com,1999:blog-8919403463715415049.post-65376811870977293262010-12-26T10:31:00.002+02:002010-12-26T10:31:32.420+02:00skin rashes
What will you do if you found skin rashes on your baby skin? Panic? Do not panic mommy.
Skin rashesin baby is one of the common baby skin symptoms on many babies. If you do not know skin rashes or diaper rashes look like, you must read my previous article which talk about What does diaper rash look like?To help you more with infant skin rashes Unknownnoreply@blogger.com1tag:blogger.com,1999:blog-8919403463715415049.post-50315867988545049612010-12-26T10:31:00.000+02:002010-12-26T10:31:10.268+02:00
What will you do if you found skin rashes on your baby skin? Panic? Do not panic mommy.
Skin rashesin baby is one of the common baby skin symptoms on many babies. If you do not know skin rashes or diaper rashes look like, you must read my previous article which talk about What does diaper rash look like?To help you more with infant skin rashes Unknownnoreply@blogger.com1tag:blogger.com,1999:blog-8919403463715415049.post-54121400243614612782010-12-26T10:25:00.000+02:002010-12-26T10:25:00.307+02:00Rashes
Rashes
atopic dermatitis (eczema): usually has its onset between two and six months with the development of itchy red areas on the cheeks, forehead, scalp, trunk and on the extensor surfaces of the arms and legs (elbows and knees). The skin may be thickened, shiny and oozing and is usually very dry. Treatment is with daily use of moisturizers and topical steroid creams during breakouts. Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-53559060835867954982010-08-02T17:41:00.002+02:002010-08-02T17:41:45.121+02:00pityriasis lichenoidesMultimedia
(Enlarge Image)Media file 1: Typical hemorrhagic crusted papules of pityriasis lichenoides et varioliformis acuta.
(Enlarge Image)Media file 2: Close-up view of typical lesions of pityriasis lichenoides et varioliformis acuta.
(Enlarge Image)Media file 3: Scaling papules of pityriasis lichenoides chronica.
(Enlarge Image)
Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-76227766306540198062010-08-02T17:40:00.002+02:002010-08-02T17:40:46.028+02:00Pityriasis LichenoidesFollow-upFurther Outpatient CarePatients with a waxing and waning course of Mucha-Habermann disease require follow-up monitoring and additional treatment depending on the severity of the disease.Regarding the questionable potential for malignant transformation, some authors have suggested that follow-up biopsy should be performed on lesions that last longer than 1 year and are refractory to Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-22011068238659532322010-08-02T17:39:00.000+02:002010-08-02T17:39:15.993+02:00pityriasis lichenoidesTreatmentMedical CareLarge ulcerations found in the febrile ulceronecrotic variant of pityriasis lichenoides et varioliformis acuta (PLEVA) require local wound care. Infected lesions may be treated with topical mupirocin and sterile dressing changes twice daily.No randomized controlled trials of the use of medications have been performed in Mucha-Habermann disease. Since the disease tends towardsUnknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-63836885426580531252010-08-02T17:38:00.000+02:002010-08-02T17:38:06.255+02:00Pityriasis LichenoidesPityriasis LichenoidesAuthor: Peter A Klein, MD, Residency Program Director, Department of Dermatology, University Hospital, State University of New York at Stony Brook
Coauthor(s): Jeffrey P Callen, MD, Professor of Medicine (Dermatology), Chief, Division of Dermatology, University of Louisville School of Medicine
Contributor Information and DisclosuresUpdated: Jul 12, 2010
Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-18136513365464980472010-08-02T17:36:00.000+02:002010-08-02T17:36:13.420+02:00Pityriasis lichenoidesPityriasis lichenoidesPityriasis lichenoides is the name given to an uncommon rash of unknown cause. The condition can range from a relatively mild chronic form to a more severe acute eruption. The mild chronic form, known as pityriasis lichenoides chronica (PLC), is characterised by the gradual development of symptomless, small, scaling papules that spontaneously flatten and regress Unknownnoreply@blogger.com1tag:blogger.com,1999:blog-8919403463715415049.post-698137574927143852010-07-23T22:29:00.036+02:002010-07-24T10:34:09.322+02:00Common rashes, Sparse Maculopapular rashes, Gonococcaemia, Lichen planus, Typhoid rose spotsSparse:
Gonococcaemia
Lichen planus
Typhoid rose spots
Flea bites
Hypopigmentation
Post-inflammation
Pityriasis alba
Pinta
Tinea versicolor Tinea versicolor, also called as Pityriasis versicolor, is one of the most common skin infections, caused by the Unknownnoreply@blogger.com7tag:blogger.com,1999:blog-8919403463715415049.post-58407540060121679962010-07-23T22:29:00.000+02:002010-07-23T22:29:21.609+02:00Common rashes, Sparse Maculopapular rashes, Gonococcaemia, Lichen planus, Typhoid rose spotsSparse:
Gonococcaemia
Lichen planus
Typhoid rose spots
Flea bites
Hypopigmentation
Post-inflammation
Pityriasis alba
Pinta
Tinea versicolor
Vitiligo
Post-kala azar dermal leishmaniasis
Leprosy
Yaws
Nodules
Onchocerciasis
Fungal infections
Erythema nodosum
Leprosy
Kaposi's sarcoma
Gout
Cutaneous leishmaniasis
Plaques/crusts
Unknownnoreply@blogger.com12tag:blogger.com,1999:blog-8919403463715415049.post-88638642648786882412010-07-22T21:40:00.007+02:002010-07-23T22:13:18.938+02:00Common rashes, Maculopapular rashes, Scabies, Rubella, Measles, Body lice, Chickenpox, Hypopigmentation, Nodules, Plaques/crusts, Urticaria, Petechiae, Vesicles, PustulesCommon rashesMaculopapular rashes Extensive:Scabies
Rubella
Measles
Body lice
Chickenpox
Secondary syphilis
Unknownnoreply@blogger.com1tag:blogger.com,1999:blog-8919403463715415049.post-58046734897089212792010-07-22T21:27:00.001+02:002010-07-22T21:35:01.904+02:00Rashes in folliculitis, shingles, vesiculation and scaling, nodule formation, syphilisRashesBasis of rashes - when looking at a rash you need to consider a few pointsThe skin varies in thickness and quantity of hair or sebaceous glands. Rashes affecting only one component of the skin will have a distribution which reflects this component (e.g. hair follicles in folliculitis or dermatomes in shingles). and Shingles looks like this
The lesions differ according toUnknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-73036577119895332162010-07-22T15:30:00.001+02:002010-07-22T15:30:41.125+02:00Virus Diseases in the Skin, Dermatology Virus diseases, Warts, exanthematous viral infectionsThere are two types of virus diseases that I see in my medical practice. The first are those caused by DNA viruses which integrate into the epidermal cell genome and behave as tumour viruses, producing verucas (WARTS). Secondly, the virus that replicate in the cell, exerting a cytolytic effect - this occurs in exanthematous viral infections
WARTS
Warts are found on the hands, feet, Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-73226438447034466792010-07-10T12:21:00.002+02:002010-07-10T12:59:36.785+02:00Hyperkeratosis, Parakeratosis, Pigmentary incontinence, Spongiosis, Vacuolar Degeneration
Hyperkeratosis: a thickening of the horny layer, usually accompanied by increase also in the granular layer. As the horny layer normally varies greatly in thickness in different sites, some experience is needed to assess minor degrees of hyperkeratosis.
Hyperkeratosis
Classification and external resources
ICD-9701.1
DiseasesDB20624
hyperkeratosis, benign,
n a nonmalignant form of a Unknownnoreply@blogger.com1tag:blogger.com,1999:blog-8919403463715415049.post-83559451433364189282010-07-10T09:36:00.000+02:002010-07-10T09:36:31.038+02:00Acantholysis, Acanthosis, Dyskeratosis,Acantholysis: Loss of cohesion between epidermal cells (keratinocytes) with consequent formation of intra-epidermal spaces containing oedema fluid and detached, rounded epithelial cells.Acanthosis: Thickening of the epidermis either focally or diffusely, due largely to increase of the stratum malpighii (Stratum Spinosum and Stratum Basali)
Dyskeratosis: Premature, Unknownnoreply@blogger.com2tag:blogger.com,1999:blog-8919403463715415049.post-89708404622031850972010-07-10T09:33:00.000+02:002010-07-10T09:33:52.944+02:00Pathology of the skin, major functions of skin, Skin Biopsy, Reactive Changes in skin disease,Inflammatory changes,Pathology of the skin is basically the same as those that occur in other organs. The changes that can be seen with the naked eye correlates well with that seen microscopically. The evolution of skin lesions can be readily followed by repeated biopsy.
The major functions of skin include sensory perception, protection against mechanical trauma, UV light and infection, insulation and Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-79743003686896235772010-07-01T16:55:00.000+02:002010-07-01T16:55:10.956+02:00Physiology and Biochemistry - FC Derm(SA) Part I18 March 2008 Paper II Physiology and Biochemistry
Question 1
Discuss the physiology of
a) Toll-like receptors. (9)
b) Tumour necrosis factor. (8)
c) Interferon. (8) [25]
Question 2
Write an essay on the short- and long-term effects of ultraviolet light on the skin. [25]
Question 3
A fire-fighter sustained burns to his face, neck, arms, and chest while trying to put out a fire in a Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-8222212196338962212010-07-01T16:49:00.000+02:002010-07-01T16:49:59.858+02:00FC Derm(SA) Part IITHE COLLEGES OF MEDICINE OF SOUTH AFRICA
Incorporated Association not for gain
Reg No 1955/000003/08
Part II of the Examination for the Fellowship of the
College of Dermatologists of South Africa
29 August 2007
Paper I Principles and Practice of Dermatology (3 hours)
All questions to be answered. Each question to be answered in a separate book (or books if more than one is
required for Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-84192474119356230392010-07-01T16:01:00.000+02:002010-07-01T16:01:38.458+02:00Principles of Pathology, Paper III1 Write notes on
a) Pathogenesis of atherosclerosis.
b) Leucocytoclastic vasculitis.
c) Dystrophic calcification. [25]
2 Discuss different mechanisms by which viruses cause disease. [25]
3 Define and discuss granulomatous inflammation. Discuss the differential diagnosis of a granulomatous inflammatory reaction in the dermis. [25]
4 a) Define metaplasia and dysplasia. Give examples of Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-68826925107092528412010-07-01T15:56:00.000+02:002010-07-01T16:05:08.611+02:00Anatomy and Histochemistry - 28 August 2007: Paper I28 August 2007: Paper I Anatomy and Histochemistry
1 Write short notes on the anatomy and histochemistry of the following organs found in skin:
a) Free nerve endings.
b) Pacinian corpuscles .
c) Meissner’s corpuscles.
d) Ruffini’s corpuscles.[25]
2 Draw an annotated diagram of
a) A desmosome-tonofilament complex.
b) A cross section of an anagen hair follicle. [25]
3 Describe the Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-8300200964669286832010-07-01T11:07:00.001+02:002010-07-01T11:07:28.279+02:00Infections of the skin and subcutaneous tissueUnknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-49457151992190047692010-07-01T10:50:00.001+02:002010-07-01T15:51:25.325+02:00Diseases of the skin and subcutaneous tissueDiseases of the skin and subcutaneous tissue
Infections of the skin and subcutaneous tissue
(L00-L08)
L00 Staphylococcal scalded skin syndrome
Pemphigus neonatorum
Ritter's disease
L01 Impetigo
L01.0 Impetigo [any organism] [Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-8258831090001436352008-03-18T22:57:00.001+02:002010-07-08T22:59:32.501+02:00Discuss the Physiology of Interferon - FC Derm(SA) Part I March 2008
InterferonFrom Wikipedia, the free encyclopedia"IFN" redirects here. For a musical album, see If'n.The molecular structure of human interferon-alphaInterferons (IFNs) are proteins made and released by lymphocytes in response to the presence of pathogens—such as viruses, bacteria, orparasites—or tumor cells. They allow communication between Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-35601217426178417502008-03-18T22:57:00.000+02:002010-07-08T22:59:02.218+02:00Discuss the Physiology of Interferon - FC Derm(SA) Part I March 2008Unknownnoreply@blogger.com0tag:blogger.com,1999:blog-8919403463715415049.post-19396566377609610332008-03-18T22:42:00.001+02:002010-07-08T22:45:21.531+02:00Discuss the Physiology of Tumor necrosis factor - FC Derm(SA) Part I March 2008
Tumor necrosis factor-alphaFrom Wikipedia, the free encyclopedia
edit
Tumor necrosis factor (TNF superfamily, member 2)
PDB rendering based on 1TNF.
[show]Available structures
Identifiers
SymbolsTNF; DIF; TNF-alpha; TNFA; TNFSF2
External IDsOMIM: 191160 MGI: 104798 HomoloGene: 496GeneCards: TNF Gene
[show]Gene Ontology
RNA expression pattern
More Unknownnoreply@blogger.com0